My point is that ALA provides adequate, not elevated levels of DHA, and there's no evidence to the contrary.
Now let's examine the actual context of my quote:
> ALA is adequately converted to DHA
I think it's worth mentioning where you make a false statement, directly after citing inefficient conversion rates in humans.
>It specifically states that ALA->DHA is only a phenomena that occurs in rats and not humans.
Which is it, inefficient or non-existent in humans?
This statement: >It is not enough to assume that ALA exerts effects through conversion to EPA and DHA, as the process is highly inefficient in humans.
... Says exactly nothing about healthy levels of DHA. It is non sequitur. It skirts the issue, it avoids the topic, the statement tries to make a huge jump in unreasonable unproven logic. This paper shows nothing but the therapeutic effect of DHA/EPA on diseased people.
There is no evidence to suggest inadequate DHA is produced from dietary ALA in otherwise healthy humans. Not in this paper, nor any I've seen.