The role of NRP1 in human sars2 infection is already well know. This study is just showing the infection in the neuron can/may/does precede it’s appearance in the serum.
Also, ACE2 is most certainly expressed in the neurons and his greatly expressed in the hippocampus.
https://www.sciencedirect.com/science/article/pii/S258900422...
The headline you used doesn't mention that this is a paper in mice, so you failed to accurately summarize one of the most important parts. So important, in fact, that it's in the title of the paper.
Edit: the link you are citing does not say what you're suggesting. "Neuroepithelium" means "epithelial cells surrounding the neurons". The paper is suggesting that they found signs of ACE2 expression in this epithelial tissue.
SAR2 is entering the neuron via NRP1 and ACE2.
They also infect wild-type mice, and see greatly reduced response in the same experiment, but claim that this is still non-zero. They hypothesize that NRP1 is mediating the infection.
The evidence for this claim is not definitive. It's an intriguing hypothesis. It's still not a paper about humans.
This is important because it can explain the neurological symptoms of Covid and possible pathway for treatment of those neurological symptoms. To anyone, like me, who had psychosis at the beginning of their Covid infection, or people who are suffering from long Covid, this is very important.
Again, this is a research paper, in mice. It has no direct relevance to any symptom you have personally experienced.
You say this like the whole story of COVID is finished.
Story for you:
https://twitter.com/JamesMelville/status/1528046141715230721
That's pretty interesting, what does this mean for anything. Like does it change how tests should work? vaccines? any mitigation measure?
I think it also shows that this is the pre-symptomatic time of the infection.
My own experiences with Covid was I had a psychotic break two days before I had a fever. I have a history of psychosis.
It’s also important to note that the Epstein-Barr virus also uses NRP1.
In my humble opinion, I feel that the destruction of NRP1 and ACE2 are responsible for many of the comorbidities we find after infection with SARS2.