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This is not more convincing because the human body produces erythritol endogenously so high erythritol blood levels may be another effect of the health problems that cause heart attacks and strokes, rather than the cause. To prove causation they would have to track each subjects consumption of erythritol rather than their blood levels.

The referenced study's methods did NOT account for the fact that overweight people are more likely to be consuming erythritol and/or on a keto diet. Highly likely to be confounding.

How does one measure "double the risk"? Does that mean if one person in a control group happened to have a stroke in that time period, and now it's two people, that counts as double? Out of 4000? Surely that can't be right.