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Syphillis makes you artistically brilliant, gondi makes you fearless, never felt so good, then just before exitus.

So, in conclusion, if the vector has requirements, the infection will try to make the host capable of fullfilment.

If the infection needs sugar, the mitochondria, will make the amobea crawl to algea.

If 30-50% of people have toxoplasma gondii, "gondi makes you fearless, never felt so good, then just before exitus" sounds like a exaggeration, because I doubt 50% are fearless, never felt so good.

Thank you for the final line, I never realised that

Its all just thesis, nothing is proven yet.

Obviously it doesn't always happen, a cold doesn't make you more sociable. On the other hand a cold virus that managed to pull that off through some freak mutation would easily outcompete all other variants.

Mitochondria are a bit special in that it's mutually beneficial, but the same could be said about the above cold virus, if the other symptoms are mild enough.

Urge to travel with corona intensifies.

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